http://stroke.ahajournals.org/cgi/content/abstract/STROKEAHA.109.570325v1
Stroke journal published a study supporting the role of erythropoietin as a neuroprotective agent in reperfusion phase of hypoxic ishemic damage.
Study demonstrates that delayed administration of EPO promotes oligodendrogenesis and attenuates white matter injury concurrently with increased neurogenesis. These effects likely contribute to the observed improvement in neurological functional outcomes.
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